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01 · ABSTRACT

Abstract

Breast cancer is the most commonly diagnosed cancer in women globally and a leading cause of cancer-related deaths. In Azerbaijan, breast cancer cases have steadily increased, with significant numbers recorded between 2020 and 2023. The human epidermal growth factor receptor 2 (HER2) gene is central in breast cancer pathology, particularly in aggressive forms with HER2 overexpression. However, emerging evidence shows that HER2 mutations may also occur in HER2-negative cases, potentially impacting prognosis and therapeutic outcomes.

Aim: This study investigates HER2 gene expression and mutation status in HER2-negative breast cancer cases and analyzes circulating matrix proteins (MMP-7, MMP-9, CYR61) in different breast cancer subtypes.

Methods: A total of 74 women aged 30–71 years with confirmed BC were enrolled. Patients were categorized as HER2-positive (n=33), HER2-negative (n=33), and triple-negative (n=8). HER2 mutation testing was conducted using AmoyDx kits via real-time PCR. Serum levels of MMP-7, MMP-9, and CYR61 were measured and statistically analyzed.

Results: No HER2 mutations were found in HER2-negative patients, supporting prior research indicating the rarity of such mutations in these subtypes. MMP-9 was significantly reduced in all breast cancer groups (p<0.001), while CYR61 levels were markedly elevated, with an inverse correlation observed between MMP-9 and CYR61 (r = -0.314, p < 0.003).

Conclusion: HER2 mutations are uncommon in HER2-negative breast cancers. Serum MMP-9 and CYR61 can serve as valuable diagnostic markers across BC subtypes. The findings support further exploration into alternative molecular targets beyond HER2 for effective treatment strategies.

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02 · PUBLICATION RECORD

Article details

JournalMedical Research Archives
IssueVol 13 No 8 (2025): Vol.13, Issue 8, August 2025
SectionResearch Articles
Published25 August 2025
DOI10.18103/mra.v13i8.6762
ISSN2375-1924
03 · RIGHTS & REUSE

Rights & reuse

This article is published under a Creative Commons Attribution License (CC BY 3.0) and may be shared or distributed by anyone as long as attribution is given to the journal.

Authors & affiliations

KL

Karimova A. Leyla

Baku State University, Baku, Azerbaijan

AG

Azizova İ. Gulnara

Azerbaijan Medical University, Baku Azerbaijan

ML

Melikova A. Leylakhanim

Azerbaijan Republic Ministry of Health National Centre of Oncology, Baku, Azerbaijan; Ministry of Science and Education, Institute of Biophysics, Baku, Azerbaijan

SI

Shahverdiyeva J. Ilaha

Azerbaijan Medical University, Baku Azerbaijan

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