↓ Read PDF
01 · ABSTRACT

Abstract

Introduction: CLX-155 is a novel oral 5’-DFCR prodrug involving 5’-DFCR as an intermediate for generating 5-FU. Unlike capecitabine, CLX-155 undergoes esterase-mediated hydrolysis in the intestinal cells rather than the liver, leading to a different metabolic and pharmacokinetic profile. This study addresses the following research questions: 1) what is the single-dose PK of CLX-155, and 2) how does it compare to capecitabine?

Methods: This study was a parallel, single-dose study with four treatment groups. Investigators randomized 48 female Balb/C mice into four treatment groups: CLX-155 at 250 mg/kg and 500 mg/kg and capecitabine at 500 mg/kg and 1000 mg/kg. Animals received oral treatment once. Investigators evaluated PK parameters via noncompartmental analysis using WinNonlin Version 7.0 (Certara, Princeton, NJ).

Results: For CLX-155, the systemic exposure (Cmax and AUC0-t) of 5-FU, 5’-DFCR, and 5’-DFUR demonstrated proportionality to the administered dose. 5’-DFCR and 5’-DFUR showed a delayed Tmax compared to 5-FU. For capecitabine, the systemic exposure (Cmax and AUC0-t) of 5-FU, 5'-DFUR, and 5’-DFCR was less than dose proportional. CLX-155 demonstrated higher exposure at 500 mg/kg compared to capecitabine at the same dose. CLX-155 displayed marginally higher 5’-DFUR and 5-FU plasma AUC0-t in relation to capecitabine at equivalent 500 mg/kg doses. CLX-155 displayed marginally higher plasma AUC0-t of 5-FU and 5’-DFUR in relation to capecitabine at the equivalent doses of 500 mg/kg.

Conclusion: CLX-155 and capecitabine experience rapid absorption following oral administration and conversion to 5’-DFCR, 5’-DFUR, and 5-FU. The results suggest the conversion of CLX-155 to its metabolites 5’-DFUR and 5-FU was more efficient than that of capecitabine. Such observations have suggested that administration of CLX-155 at a lower dose level is a possibility. CLX-155’s infusion-like conversion to its metabolites 5’-DFUR and 5-FU provided a unique PK profile that may explain its antitumor activity in animals at half the dose of capecitabine reported in the previous study.

↓ Read PDF
02 · PUBLICATION RECORD

Article details

JournalMedical Research Archives
IssueVol 12 No 9 (2024): Vol 12 No 9 (2024): September ISSUE, Issue 9, VOl.12
SectionResearch Articles
Published05 October 2024
DOI10.18103/mra.v12i9.5709
ISSN2375-1924
03 · RIGHTS & REUSE

Rights & reuse

This article is published under a Creative Commons Attribution License (CC BY 3.0) and may be shared or distributed by anyone as long as attribution is given to the journal.

Authors & affiliations

JY

John M. York, PharmD, MBA, PhDc (Dr. Y)

Ernest Mario School of Pharmacy, Rutgers, the State University of New Jersey, Piscataway, NJ; Institute for the Global Entrepreneur at the Rady School of Management and the Jacobs School of Engineering, University of California, San Diego, CA; Burnett School of Medicine, Texas Christian University, Ft. Worth, Texas.

SK

Sophie Kang

Ernest Mario School of Pharmacy, Rutgers, the State University of New Jersey, Piscataway, NJ.

AD

Ava Dalton

Ernest Mario School of Pharmacy, Rutgers, the State University of New Jersey, Piscataway, NJ.

YT

Yearam Tak

Ernest Mario School of Pharmacy, Rutgers, the State University of New Jersey, Piscataway, NJ.

NB

Natasha Boyette

Ernest Mario School of Pharmacy, Rutgers, the State University of New Jersey, Piscataway, NJ.

MK

Mahesh Kandula

Cellix Biosciences, Inc., Newark, New Jersey;  Cellix Bio Private Limited, Hyderabad, India.

SA

Subbu Apparsundaram

Cellix Biosciences, Inc., Newark, New Jersey; Cellix Bio Private Limited, Hyderabad, India.

Medical Research Archives

Submit your own article

Register as an author to reserve your spot in the next issue of the Medical Research Archives.

Start your submission  ↗