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01 · ABSTRACT

Abstract

The lifespans of mammalian species vary across orders of magnitude, with the shortest on the order of a year and the longest more than one hundred years. These lifespans are influenced by both genetic and environmental factors. Here we asked whether we can define a molecular process that can be used to define the intrinsic molecular lifespan of a species that is largely independent of environmental factors. To address this question, we have focused on ‘epigenetic clocks’ - highly accurate age-predicting biomarkers based on DNA methylation. Our previous research has demonstrated that the alterations in DNA methylation related to age are non-linear, changing rapidly early in life and slowing down with advancing age. We have proposed the use of saturating exponential functions to represent these changes, which tend to stabilize at terminal methylation levels towards the end of an organism's lifespan. Our current study expands upon this by examining the exponential aging timescales across various mammalian species. We show that the DNA methylation trajectories of a broad range of species, ranging from mice to humans, adhere to a saturating exponential function. Furthermore, we find the timescale of this exponential decay to be about one third of a species’ lifespan. This striking and novel observation implies that we can define an intrinsic molecular lifespan of a species that is largely unaffected by environmental factors. Although the exact mechanisms behind the variation in species-specific rates remain unclear, we hypothesize that they may be linked to the distinct metabolic rates found in each species.

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02 · PUBLICATION RECORD

Article details

JournalMedical Research Archives
IssueVol 12 No 8 (2024): Vol 12 No 8 (2024): August ISSUE, Issue 8, VOl.12
SectionResearch Articles
Published29 August 2024
DOI10.18103/mra.v12i8.5560
ISSN2375-1924
03 · RIGHTS & REUSE

Rights & reuse

This article is published under a Creative Commons Attribution License (CC BY 3.0) and may be shared or distributed by anyone as long as attribution is given to the journal.

Authors & affiliations

PR

Prashansa Ratan

Department of Pediatrics, David Geffen School of Medicine, University of California Los Angeles, California, USA.

SD

Sherin U. Devaskar

Department of Pediatrics, David Geffen School of Medicine, University of California Los Angeles, California, USA.

MP

Matteo Pellegrini

Department of Molecular, Cell and Developmental Biology, University of California Los Angeles, California, USA.

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